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  • Alpha-Synuclein Aggregation in Parkinsons Disease - Frontiers
    Introduction Parkinson's disease (PD) is the second most common neurodegenerative disorder that falls under the category of synucleinopathy () PD is characterized by distinct aging-independent loss of dopaminergic neurons in substantia nigra pars compacta (SNpc) region and the decrease in dopamine levels () Possibly, PD leads to the loss of terminal ends of striatum, which occurs before the
  • A detailed review of pathophysiology, epidemiology, cellular and . . .
    Degradation of neurons is triggered by gene mutations that encode for central nervous system (CNS) proteins In particular, SNCA (alpha-synuclein protein) turns self-aggregates and abnormal This inflexible alpha-synuclein is a crucial element of LB, the cellular accumulation that characterizes PD (Sun and Armstrong 2021) Atypical protein-disrupting systems, like the ubiquitin–proteasome
  • Alpha-Synuclein Pathophysiology in Neurodegenerative Disorders: A . . .
    Worldwide aging has contributed to the growth of prevalence of neurodegenerative diseases (NDDs), including Parkinson’s disease among the elderlies The advanced destruction of dopaminergic neurons in the substantia nigra, due to many accelerator factors in the brain is the main mechanism of Parkinson’s disease The pathological aggregated alpha-synuclein (α-syn), a protein implicated in
  • Role of dopamine in the pathophysiology of Parkinson’s disease - Springer
    A pathological feature of Parkinson’s disease (PD) is the progressive loss of dopaminergic neurons and decreased dopamine (DA) content in the substantia nigra pars compacta in PD brains DA is the neurotransmitter of dopaminergic neurons Accumulating evidence suggests that DA interacts with environmental and genetic factors to contribute to PD pathophysiology Disturbances of DA synthesis
  • Novel approaches targeting α-Synuclein for Parkinsons Disease: Current . . .
    Parkinson's Disease (PD) is the second most common neurodegenerative condition characterized by the progressive loss of dopamine-producing neurons in the substantia nigra pars compacta (SNpc) and the degeneration of multiple motor and nonmotor basal ganglia circuits [1, 2] This loss of nigrostriatal dopaminergic innervation is central to the disease's pathogenesis, which may also involve cells
  • Parkinsons disease - The Lancet
    Parkinson's disease is a recognisable clinical syndrome with a range of causes and clinical presentations Parkinson's disease represents a fast-growing neurodegenerative condition; the rising prevalence worldwide resembles the many characteristics typically observed during a pandemic, except for an infectious cause In most populations, 3–5% of Parkinson's disease is explained by genetic
  • Interactions of dopamine, iron, and alpha-synuclein linked to . . .
    Dopamine metabolism, alpha-synuclein pathology, and iron homeostasis have all been implicated as potential contributors to the unique vulnerability of substantia nigra dopaminergic neurons which preferentially decline in Parkinson's disease and some rare neurodegenerative disorders with shared pathological features
  • Alpha-Synuclein Pathophysiology in Neurodegenerative Disorders: A . . .
    Keywords Neurodegenerative diseases · Parkinson’s disease · Alpha-synuclein · Alpha- synucleinopathy Background Nowadays, among the general population, aging in aging is a critical concern (United Nations 2023) The risk of develop-ing age-related neurodegenerative diseases (NDDs), such as Parkinson’s disease, increases in the elderly





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